Cognitive Communication Disorders – Michael L Kimbarow

This is a good first step, but careful consideration of the treatments and measurement of outcomes are needed. There are very few direct comparisons between TBI and RHD groups, and so while many of the deficits appear similar, it is not clear whether they may differ in terms of response to treatment. Clinicians must carefully review the literature, examine the strength of the existing treatment studies, and then determine whether or not a specific treatment may be appropriate for any one client with RHD.

The following six questions (Cicerone, 2005; based on Sackett, Straus, Richardson, Rosenberg, & Haynes, 2000) are used as a framework for guiding the selection of treatments for individuals with RHD. Table 4–6 provides a direct comparison between a pragmatic treatment designed for young adults with TBI and a hypothetical client with RHD. 1. Is my client sufficiently similar, in most important ways, to those described in the treatment study? The clinician must determine what factors are important to consider. These may include age, etiology, location, acute versus chronic stage, and degenerative condition.

When using treatments designed for other groups, obviously the etiology will differ. The location and extent of lesion(s) also may differ, given that most clients with RHD have damage due to a focal stroke while TBI generally results in diffuse damage (in addition to focal damage in some cases). Age often will differ between these groups, as young adults are most susceptible to TBIs, and strokes occur most often in older adults.

The course of both etiologies is stable or improving (i.e., neither is degenerative). Whether a client is in acute versus chronic stages should be considered. In acute stages, the potential for spontaneous recovery exists. In chronic stages, if clients have had negative experiences due to their disorders (e.g., a loss of friends due to pragmatic deficits), then they may be more motivated to seek out treatment and participate in therapy, as opposed to those in acute stages who do not have a clear understanding of the implications of their deficits.

2. Is the nature of my client’s cognitive impairment similar to that targeted in the treatment research? As mentioned above, deficits in attention, memory, executive function, and discourse/pragmatics have been reported to be similar across TBI and RHD groups. Again, one should be careful with these comparisons as they have not been carefully evaluated to determine the extent to which the symptoms actually do overlap.

Copyright © 2021 by Plural Publishing, Inc. Typeset in 10.5/13 Palatino by Flanagan’s Publishing Services, Inc. Printed in the United States of America by McNaughton & Gunn, Inc. All rights, including that of translation, reserved. No part of this publication may be reproduced, stored in a retrieval system, or transmitted in any form or by any means, electronic, mechanical, recording, or otherwise, including photocopying, recording, taping, Web distribution, or information storage and retrieval systems without the prior written consent of the publisher.

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Disclaimer: Please note that ancillary content (such as documents, audio, and video, etc.) may not be included as published in the original print version of this book. Library of Congress Cataloging-in-Publication Data Names: Kimbarow, Michael L., 1953– editor. Title: Cognitive communication disorders / [edited by] Michael L. Kimbarow. Description: Third edition. | San Diego, CA : Plural, [2021] | Includes bibliographical references and index. Identifiers: LCCN 2019011615| ISBN 9781635501568 (alk.

paper) | ISBN 1635501563 (alk. paper) Subjects: | MESH: Cognition Disorders—complications | Communication Disorders—etiology Classification: LCC RD594 | NLM WM 204 | DDC 617.4/81044—dc23 LC record available at https://lccn.loc.gov/2019011615 1kitap1.com/en Contents Preface Acknowledgments Contributors 1. Attention Sarah Villard 2. Principles of Human Memory: An Integrative Clinical Neuroscience Perspective Fofi Constantinidou 3. Executive Functions: Theory, Assessment, and Treatment Mary H. Purdy 4. Cognitive Communication Deficits Associated With Right Hemisphere Brain Damage Margaret Lehman Blake 5.

Primary Progressive Aphasia Heather Dial and Maya Henry 6. Dementia: Concepts and Contemporary Practice Nidhi Mahendra 7. Cognitive Communication Disorders of Mild Traumatic Brain Injury Carole Roth and Kathryn Hardin 8. Traumatic Brain Injury Jessica A. Brown, Sarah E. Wallace, and Michael L. Kimbarow Index 1kitap1.com/en Preface Welcome to the third edition of Cognitive Communication Disorders. As I write this, it’s hard to fathom that five years have gone by since the second edition was published and almost 10 years have elapsed since the inaugural release of the book in 2011.

This is a short excerpt from the opening of “” by Unknown, quoted for review and introduction purposes. All rights belong to the copyright holders.

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